Diabetes MellitusJohn A. Galloway, Janet H. Potvin, Charles R. Shuman Lilly Research Laboratories, 1988 - 335 pages |
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Page 47
... Figure 4.2 . Synthesis of insulin within islet B cells. Figure 4.1 . INTERSTITIAL SPACE Glucose Transport Membrane Membrane Effects Insulin -RECEPTOR α α B B ATP - ADP Tyrosine Kinase Enzymes INTRACELLULAR SPACE Inactivated Abbreviations ...
... Figure 4.2 . Synthesis of insulin within islet B cells. Figure 4.1 . INTERSTITIAL SPACE Glucose Transport Membrane Membrane Effects Insulin -RECEPTOR α α B B ATP - ADP Tyrosine Kinase Enzymes INTRACELLULAR SPACE Inactivated Abbreviations ...
Page 141
... Figure 8.1 . LIVER Increased Glucose Production Receptor + Postreceptor Defect GLUCOSE + MUSCLE PANCREAS Impaired Insulin Secretion The early - phase insulin response , an insulin pulse that appears within three minutes of the glucose ...
... Figure 8.1 . LIVER Increased Glucose Production Receptor + Postreceptor Defect GLUCOSE + MUSCLE PANCREAS Impaired Insulin Secretion The early - phase insulin response , an insulin pulse that appears within three minutes of the glucose ...
Page 234
... Figure 13.6 . Insulin Effect Morning Afternoon Evening Night Regular insulin Regular insulin NPH / Lente insulin NPH / Lente insulin ↑ ↑ ↑ Breakfast Lunch Time insulin Insulin administered type Presupper or Prebedtime NPH Meals ...
... Figure 13.6 . Insulin Effect Morning Afternoon Evening Night Regular insulin Regular insulin NPH / Lente insulin NPH / Lente insulin ↑ ↑ ↑ Breakfast Lunch Time insulin Insulin administered type Presupper or Prebedtime NPH Meals ...
Contents
Definition | 1 |
Chapter 2 iabetes Mellitus and Heredity | 15 |
Chapter 3 athophysiology of Diabetes Mellitus | 27 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormalities action activity agents antibodies associated betes blood glucose blood glucose concentrations body carbohydrate cause cells changes Chapter clinical complications continuous daily decrease determinations diabetes mellitus diabetic diet diabetic ketoacidosis diagnosis diet disease dose early effect exercise factors Figure given glucose tolerance glycemic glycosylated hemoglobin hormone human insulin hypoglycemia impaired important improved increased indicated individuals initial injection insulin infusion insulin receptor insulin resistance insulin secretion insulin therapy insulin-dependent levels lipoprotein liver meals measure metabolic methods mg/dl mmol/L monitoring muscle myocardial infarction needed normal obese occur oral patients with diabetes patients with Type plasma glucose pregnancy prevent production protein pump receptor reduced regular renal reported response retinopathy risk severe Source studies subcutaneous subjects Table therapy tion tissue treated treatment Type I diabetes Type II units urine usually values weight