Diabetes MellitusJohn A. Galloway, Janet H. Potvin, Charles R. Shuman Lilly Research Laboratories, 1988 - 335 pages |
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Page 70
... VLDL particles resembles that of chylomicrons ( Figure 5.3 ) . However , VLDL par- ticles are formed in the liver , contain apo B - 100 , and are precursors in part to both IDL and LDL . The distinction between chylomicrons and VLDL is ...
... VLDL particles resembles that of chylomicrons ( Figure 5.3 ) . However , VLDL par- ticles are formed in the liver , contain apo B - 100 , and are precursors in part to both IDL and LDL . The distinction between chylomicrons and VLDL is ...
Page 71
... VLDL metabolism . VLDL are formed in the liver from triglyceride and apo B - 100 . The hydrolysis of VLDL triglyceride shares the apo CII - LPL mechanism with chylomicrons . Resultant IDL con- tains more apo E and more cholesterol ester ...
... VLDL metabolism . VLDL are formed in the liver from triglyceride and apo B - 100 . The hydrolysis of VLDL triglyceride shares the apo CII - LPL mechanism with chylomicrons . Resultant IDL con- tains more apo E and more cholesterol ester ...
Page 74
... VLDL , IDL , and LDL . Abnormal HDL . Apo CII deficiency 1 : 106 Enzyme Disorders LPL deficiency LCAT deficiency 1 : 105 1 : 106 Elevation of VLDL and chylomicrons . Elevated chylomicrons and VLDL . Abnormal composition of all ...
... VLDL , IDL , and LDL . Abnormal HDL . Apo CII deficiency 1 : 106 Enzyme Disorders LPL deficiency LCAT deficiency 1 : 105 1 : 106 Elevation of VLDL and chylomicrons . Elevated chylomicrons and VLDL . Abnormal composition of all ...
Contents
Definition | 1 |
Chapter 2 iabetes Mellitus and Heredity | 15 |
Chapter 3 athophysiology of Diabetes Mellitus | 27 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormalities action activity agents antibodies associated betes blood glucose blood glucose concentrations body carbohydrate cause cells changes Chapter clinical complications continuous daily decrease determinations diabetes mellitus diabetic diet diabetic ketoacidosis diagnosis diet disease dose early effect exercise factors Figure given glucose tolerance glycemic glycosylated hemoglobin hormone human insulin hypoglycemia impaired important improved increased indicated individuals initial injection insulin infusion insulin receptor insulin resistance insulin secretion insulin therapy insulin-dependent levels lipoprotein liver meals measure metabolic methods mg/dl mmol/L monitoring muscle myocardial infarction needed normal obese occur oral patients with diabetes patients with Type plasma glucose pregnancy prevent production protein pump receptor reduced regular renal reported response retinopathy risk severe Source studies subcutaneous subjects Table therapy tion tissue treated treatment Type I diabetes Type II units urine usually values weight