Diabetes MellitusJohn A. Galloway, Janet H. Potvin, Charles R. Shuman Lilly Research Laboratories, 1988 - 335 pages |
From inside the book
Results 1-3 of 91
Page 28
... cells in their pan- creatic islets , with preservation of the glucagon- secreting a cells and the somatostatin - secreting A cells . Although hereditary factors are associ- ated with the development of Type I diabetes , it is the ...
... cells in their pan- creatic islets , with preservation of the glucagon- secreting a cells and the somatostatin - secreting A cells . Although hereditary factors are associ- ated with the development of Type I diabetes , it is the ...
Page 70
... cells from absorbed triglyceride ( Figure 5.2 ) . Apo B - 48 , which gives structure to the chylomicron , is necessary for transport of the par- ticle through the cellular membrane to the lymph vessel and stays with the chylomicron ...
... cells from absorbed triglyceride ( Figure 5.2 ) . Apo B - 48 , which gives structure to the chylomicron , is necessary for transport of the par- ticle through the cellular membrane to the lymph vessel and stays with the chylomicron ...
Page 191
... cells Rats None One injection anti- lymphocyte serum Rats None Cyclosporine A 3 days Rats None Cyclosporine A 3 days ... cells or a foreign class II anti- gen , the transplanted tissue is not rejected . Suc- cessful transplantation of ...
... cells Rats None One injection anti- lymphocyte serum Rats None Cyclosporine A 3 days Rats None Cyclosporine A 3 days ... cells or a foreign class II anti- gen , the transplanted tissue is not rejected . Suc- cessful transplantation of ...
Contents
Definition | 1 |
Chapter 2 iabetes Mellitus and Heredity | 15 |
Chapter 3 athophysiology of Diabetes Mellitus | 27 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormalities action activity agents antibodies associated betes blood glucose blood glucose concentrations body carbohydrate cause cells changes Chapter clinical complications continuous daily decrease determinations diabetes mellitus diabetic diet diabetic ketoacidosis diagnosis diet disease dose early effect exercise factors Figure given glucose tolerance glycemic glycosylated hemoglobin hormone human insulin hypoglycemia impaired important improved increased indicated individuals initial injection insulin infusion insulin receptor insulin resistance insulin secretion insulin therapy insulin-dependent levels lipoprotein liver meals measure metabolic methods mg/dl mmol/L monitoring muscle myocardial infarction needed normal obese occur oral patients with diabetes patients with Type plasma glucose pregnancy prevent production protein pump receptor reduced regular renal reported response retinopathy risk severe Source studies subcutaneous subjects Table therapy tion tissue treated treatment Type I diabetes Type II units urine usually values weight