Diabetes MellitusJohn A. Galloway, Janet H. Potvin, Charles R. Shuman Lilly Research Laboratories, 1988 - 335 pages |
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Page 72
... cholesterol transport or centripetal cholesterol transport . Nascent HDL acquire unesterified cholesterol from all cell surfaces ( whether nascent HDL can also acquire cholesterol from atherosclerotic plaques remains uncertain ) . The ...
... cholesterol transport or centripetal cholesterol transport . Nascent HDL acquire unesterified cholesterol from all cell surfaces ( whether nascent HDL can also acquire cholesterol from atherosclerotic plaques remains uncertain ) . The ...
Page 73
... cholesterol transport pathway . Nascent HDL are formed in the intestine , liver , and blood from surplus chylomicron surface . These particles take up unesterified cholesterol from peripheral cells ( and although unknown at this time ...
... cholesterol transport pathway . Nascent HDL are formed in the intestine , liver , and blood from surplus chylomicron surface . These particles take up unesterified cholesterol from peripheral cells ( and although unknown at this time ...
Page 76
... cholesterol ( and thus LDL cholesterol ) concentra- tions ; and decreased , normal , or increased HDL cholesterol concentrations . However , a distinc- tion should be made between different groups of patients with diabetes ...
... cholesterol ( and thus LDL cholesterol ) concentra- tions ; and decreased , normal , or increased HDL cholesterol concentrations . However , a distinc- tion should be made between different groups of patients with diabetes ...
Contents
Definition | 1 |
Chapter 2 iabetes Mellitus and Heredity | 15 |
Chapter 3 athophysiology of Diabetes Mellitus | 27 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormalities action activity agents antibodies associated betes blood glucose blood glucose concentrations body carbohydrate cause cells changes Chapter clinical complications continuous daily decrease determinations diabetes mellitus diabetic diet diabetic ketoacidosis diagnosis diet disease dose early effect exercise factors Figure given glucose tolerance glycemic glycosylated hemoglobin hormone human insulin hypoglycemia impaired important improved increased indicated individuals initial injection insulin infusion insulin receptor insulin resistance insulin secretion insulin therapy insulin-dependent levels lipoprotein liver meals measure metabolic methods mg/dl mmol/L monitoring muscle myocardial infarction needed normal obese occur oral patients with diabetes patients with Type plasma glucose pregnancy prevent production protein pump receptor reduced regular renal reported response retinopathy risk severe Source studies subcutaneous subjects Table therapy tion tissue treated treatment Type I diabetes Type II units urine usually values weight