Diabetes MellitusJohn A. Galloway, Janet H. Potvin, Charles R. Shuman Lilly Research Laboratories, 1988 - 335 pages |
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Page 68
... Chylomicron remnants , IDL Figure 5.2 . Chylomicron metabolism . Chylomicrons are formed in. Table 5.1 . Apoproteins and Enzymes in Lipid Transport Apoproteins A I , II , IV B - 100 B - 48 To Be Found in HDL , Chylomicron VLDL , IDL ...
... Chylomicron remnants , IDL Figure 5.2 . Chylomicron metabolism . Chylomicrons are formed in. Table 5.1 . Apoproteins and Enzymes in Lipid Transport Apoproteins A I , II , IV B - 100 B - 48 To Be Found in HDL , Chylomicron VLDL , IDL ...
Page 69
... Chylomicron metabolism . Chylomicrons are formed in intestinal cells from absorbed fat and apo B48 . Apo AI and AIV are loosely attached . After secretion into the lymph and transport to the blood , apo AI is exchanged with apo C from ...
... Chylomicron metabolism . Chylomicrons are formed in intestinal cells from absorbed fat and apo B48 . Apo AI and AIV are loosely attached . After secretion into the lymph and transport to the blood , apo AI is exchanged with apo C from ...
Page 70
... chylomicron during its transport in the plasma . Other apoproteins added to the chylomicron in the mucosal cells are apo AI and apo AIV . Changes in chylomicron composition occur soon after the particle enters the lymph vessel : surface ...
... chylomicron during its transport in the plasma . Other apoproteins added to the chylomicron in the mucosal cells are apo AI and apo AIV . Changes in chylomicron composition occur soon after the particle enters the lymph vessel : surface ...
Contents
Definition | 1 |
Chapter 2 iabetes Mellitus and Heredity | 15 |
Chapter 3 athophysiology of Diabetes Mellitus | 27 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormalities action activity agents antibodies associated betes blood glucose blood glucose concentrations body carbohydrate cause cells changes Chapter clinical complications continuous daily decrease determinations diabetes mellitus diabetic diet diabetic ketoacidosis diagnosis diet disease dose early effect exercise factors Figure given glucose tolerance glycemic glycosylated hemoglobin hormone human insulin hypoglycemia impaired important improved increased indicated individuals initial injection insulin infusion insulin receptor insulin resistance insulin secretion insulin therapy insulin-dependent levels lipoprotein liver meals measure metabolic methods mg/dl mmol/L monitoring muscle myocardial infarction needed normal obese occur oral patients with diabetes patients with Type plasma glucose pregnancy prevent production protein pump receptor reduced regular renal reported response retinopathy risk severe Source studies subcutaneous subjects Table therapy tion tissue treated treatment Type I diabetes Type II units urine usually values weight