Diabetes MellitusJohn A. Galloway, Janet H. Potvin, Charles R. Shuman Lilly Research Laboratories, 1988 - 335 pages |
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Page 166
... Glycemic control , assessed by glycosylated hemoglobin ( HbA , ) values , in relation to number of daily SMBG determinations . After one year of intensive therapy , HbA , values were near normal ( “ Initial ” ) . Patients had been per ...
... Glycemic control , assessed by glycosylated hemoglobin ( HbA , ) values , in relation to number of daily SMBG determinations . After one year of intensive therapy , HbA , values were near normal ( “ Initial ” ) . Patients had been per ...
Page 167
... glycemic excursions , while glycosylated hemoglobin docu- ments average glycemic control . SMBG obviates the disadvantages of urine glucose monitoring , which may be misleading and inadequate and correlates poorly with glycemia . Up to ...
... glycemic excursions , while glycosylated hemoglobin docu- ments average glycemic control . SMBG obviates the disadvantages of urine glucose monitoring , which may be misleading and inadequate and correlates poorly with glycemia . Up to ...
Page 170
... glycemic control during the lifespan of the hemoglobin molecule . The correlations are strongest for glycemic control four to eight weeks before glycosylated hemoglobin determination . Values appear to be more sensitive to deteriora ...
... glycemic control during the lifespan of the hemoglobin molecule . The correlations are strongest for glycemic control four to eight weeks before glycosylated hemoglobin determination . Values appear to be more sensitive to deteriora ...
Contents
Definition | 1 |
Chapter 2 iabetes Mellitus and Heredity | 15 |
Chapter 3 athophysiology of Diabetes Mellitus | 27 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormalities action activity agents antibodies associated betes blood glucose blood glucose concentrations body carbohydrate cause cells changes Chapter clinical complications continuous daily decrease determinations diabetes mellitus diabetic diet diabetic ketoacidosis diagnosis diet disease dose early effect exercise factors Figure given glucose tolerance glycemic glycosylated hemoglobin hormone human insulin hypoglycemia impaired important improved increased indicated individuals initial injection insulin infusion insulin receptor insulin resistance insulin secretion insulin therapy insulin-dependent levels lipoprotein liver meals measure metabolic methods mg/dl mmol/L monitoring muscle myocardial infarction needed normal obese occur oral patients with diabetes patients with Type plasma glucose pregnancy prevent production protein pump receptor reduced regular renal reported response retinopathy risk severe Source studies subcutaneous subjects Table therapy tion tissue treated treatment Type I diabetes Type II units urine usually values weight