Diabetes MellitusJohn A. Galloway, Janet H. Potvin, Charles R. Shuman Lilly Research Laboratories, 1988 - 335 pages |
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Page 31
... Glucose Glucose Glycerol FFA FFA ADIPOCYTE BOARD OF DIRECTORS LIVER , INC . Glucagon ACTH Insulin PANCREATIC ISLETS ... hepatic portal circulation . Digestion and absorp- tion of carbohydrates are not altered significantly in patients ...
... Glucose Glucose Glycerol FFA FFA ADIPOCYTE BOARD OF DIRECTORS LIVER , INC . Glucagon ACTH Insulin PANCREATIC ISLETS ... hepatic portal circulation . Digestion and absorp- tion of carbohydrates are not altered significantly in patients ...
Page 145
... glucose disposal rate seen in patients with Type II diabetes correlated with increased fasting blood glucose concentrations . Excessive Glucose Production by the Liver 10 Glucose clamp studies usually measure hepatic glucose output as ...
... glucose disposal rate seen in patients with Type II diabetes correlated with increased fasting blood glucose concentrations . Excessive Glucose Production by the Liver 10 Glucose clamp studies usually measure hepatic glucose output as ...
Page 146
... Glucose uptake by cells ( Muscle and Prolonged hyper- glycemia adipose tissue ) Beta cell exhaustion 1 ° Liver defect- Hepatic glucose uptake Prolonged hyper- glycemia 1 ° Beta - cell defect Glucose - mediated insulin secretion ...
... Glucose uptake by cells ( Muscle and Prolonged hyper- glycemia adipose tissue ) Beta cell exhaustion 1 ° Liver defect- Hepatic glucose uptake Prolonged hyper- glycemia 1 ° Beta - cell defect Glucose - mediated insulin secretion ...
Contents
Definition | 1 |
Chapter 2 iabetes Mellitus and Heredity | 15 |
Chapter 3 athophysiology of Diabetes Mellitus | 27 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormalities action activity agents antibodies associated betes blood glucose blood glucose concentrations body carbohydrate cause cells changes Chapter clinical complications continuous daily decrease determinations diabetes mellitus diabetic diet diabetic ketoacidosis diagnosis diet disease dose early effect exercise factors Figure given glucose tolerance glycemic glycosylated hemoglobin hormone human insulin hypoglycemia impaired important improved increased indicated individuals initial injection insulin infusion insulin receptor insulin resistance insulin secretion insulin therapy insulin-dependent levels lipoprotein liver meals measure metabolic methods mg/dl mmol/L monitoring muscle myocardial infarction needed normal obese occur oral patients with diabetes patients with Type plasma glucose pregnancy prevent production protein pump receptor reduced regular renal reported response retinopathy risk severe Source studies subcutaneous subjects Table therapy tion tissue treated treatment Type I diabetes Type II units urine usually values weight