Diabetes MellitusJohn A. Galloway, Janet H. Potvin, Charles R. Shuman Lilly Research Laboratories, 1988 - 335 pages |
From inside the book
Results 1-3 of 65
Page 46
... hormones ( glucagon , epinephrine , growth hormone , and cortisol ) which antagonize insulin action , and tissue response to these hormones . Imbalances can lead to hypoglycemia ( e.g. , excess secretion of insulin by an insulinoma ) or ...
... hormones ( glucagon , epinephrine , growth hormone , and cortisol ) which antagonize insulin action , and tissue response to these hormones . Imbalances can lead to hypoglycemia ( e.g. , excess secretion of insulin by an insulinoma ) or ...
Page 58
... Hormone In addition to affecting growth by promoting secretion of insulinlike peptides called somatome- dins , growth hormone figures importantly in sub- strate homeostasis . Like cortisol , its secretion is pulsatile and is increased ...
... Hormone In addition to affecting growth by promoting secretion of insulinlike peptides called somatome- dins , growth hormone figures importantly in sub- strate homeostasis . Like cortisol , its secretion is pulsatile and is increased ...
Page 61
... hormone excess and unstable glycemic control . Present evidence indicates that epinephrine and growth hormone may be the major counterregulatory hormones involved in this phenomenon . Dawn Phenomenon The Somogyi effect should be ...
... hormone excess and unstable glycemic control . Present evidence indicates that epinephrine and growth hormone may be the major counterregulatory hormones involved in this phenomenon . Dawn Phenomenon The Somogyi effect should be ...
Contents
Definition | 1 |
Chapter 2 iabetes Mellitus and Heredity | 15 |
Chapter 3 athophysiology of Diabetes Mellitus | 27 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormalities action activity agents antibodies associated betes blood glucose blood glucose concentrations body carbohydrate cause cells changes Chapter clinical complications continuous daily decrease determinations diabetes mellitus diabetic diet diabetic ketoacidosis diagnosis diet disease dose early effect exercise factors Figure given glucose tolerance glycemic glycosylated hemoglobin hormone human insulin hypoglycemia impaired important improved increased indicated individuals initial injection insulin infusion insulin receptor insulin resistance insulin secretion insulin therapy insulin-dependent levels lipoprotein liver meals measure metabolic methods mg/dl mmol/L monitoring muscle myocardial infarction needed normal obese occur oral patients with diabetes patients with Type plasma glucose pregnancy prevent production protein pump receptor reduced regular renal reported response retinopathy risk severe Source studies subcutaneous subjects Table therapy tion tissue treated treatment Type I diabetes Type II units urine usually values weight