Diabetes MellitusJohn A. Galloway, Janet H. Potvin, Charles R. Shuman Lilly Research Laboratories, 1988 - 335 pages |
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Page 34
... increased in uncontrolled diabe- tes . They were surprised to find , however , that protein synthesis was also increased . Protein breakdown increased more than protein synthe- sis , resulting in a net loss of protein in the patients ...
... increased in uncontrolled diabe- tes . They were surprised to find , however , that protein synthesis was also increased . Protein breakdown increased more than protein synthe- sis , resulting in a net loss of protein in the patients ...
Page 39
... increased in muscle cells from the free fatty acid generated locally from muscle triglyceride hydro- lysis , and from free fatty acid transported in the blood from adipose tissue lipolysis . The muscle metabolizes the free fatty acid to ...
... increased in muscle cells from the free fatty acid generated locally from muscle triglyceride hydro- lysis , and from free fatty acid transported in the blood from adipose tissue lipolysis . The muscle metabolizes the free fatty acid to ...
Page 40
... increased micro- and macroangiopathy in subjects with diabetes with hyperglycemia : increased glycoprotein synthesis ; increased glycosylated protein synthesis ; increased sorbitol and fructose production ; and increased platelet ...
... increased micro- and macroangiopathy in subjects with diabetes with hyperglycemia : increased glycoprotein synthesis ; increased glycosylated protein synthesis ; increased sorbitol and fructose production ; and increased platelet ...
Contents
Definition | 1 |
Chapter 2 iabetes Mellitus and Heredity | 15 |
Chapter 3 athophysiology of Diabetes Mellitus | 27 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormalities action activity agents antibodies associated betes blood glucose blood glucose concentrations body carbohydrate cause cells changes Chapter clinical complications continuous daily decrease determinations diabetes mellitus diabetic diet diabetic ketoacidosis diagnosis diet disease dose early effect exercise factors Figure given glucose tolerance glycemic glycosylated hemoglobin hormone human insulin hypoglycemia impaired important improved increased indicated individuals initial injection insulin infusion insulin receptor insulin resistance insulin secretion insulin therapy insulin-dependent levels lipoprotein liver meals measure metabolic methods mg/dl mmol/L monitoring muscle myocardial infarction needed normal obese occur oral patients with diabetes patients with Type plasma glucose pregnancy prevent production protein pump receptor reduced regular renal reported response retinopathy risk severe Source studies subcutaneous subjects Table therapy tion tissue treated treatment Type I diabetes Type II units urine usually values weight