Diabetes MellitusJohn A. Galloway, Janet H. Potvin, Charles R. Shuman Lilly Research Laboratories, 1988 - 335 pages |
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Page 254
... ketone body , 3 - hydroxybutyrate . Occasionally an apparently negative test for ketones results when there is in fact a predominantly 3 - hydroxybutyric acidosis . Blood testing is emphasized more than urine testing because in an ...
... ketone body , 3 - hydroxybutyrate . Occasionally an apparently negative test for ketones results when there is in fact a predominantly 3 - hydroxybutyric acidosis . Blood testing is emphasized more than urine testing because in an ...
Page 261
... ketone bodies . While the ketone bodies are not of themselves harmful , marked accumulation of hydrogen ions causes acidemia ( both major ketone bodies are weak acids 99 % dissociated at physiologic pH ) , which in turn stimulates ...
... ketone bodies . While the ketone bodies are not of themselves harmful , marked accumulation of hydrogen ions causes acidemia ( both major ketone bodies are weak acids 99 % dissociated at physiologic pH ) , which in turn stimulates ...
Page 267
... ketone bodies ( mmol / L ) 0.5-2.0 3.0-27.0 Blood lactate ( mmol / L ) 1.0-6.0 0.8-4.0 BUN ( mg / dl ) 30-80 15-40 Plasma osmolality ( mOsm / L ) 340-450 310-380 Fluid deficiency ( L ) 6-12 3-7 " Mean given in brackets . is eased ...
... ketone bodies ( mmol / L ) 0.5-2.0 3.0-27.0 Blood lactate ( mmol / L ) 1.0-6.0 0.8-4.0 BUN ( mg / dl ) 30-80 15-40 Plasma osmolality ( mOsm / L ) 340-450 310-380 Fluid deficiency ( L ) 6-12 3-7 " Mean given in brackets . is eased ...
Contents
Definition | 1 |
Chapter 2 iabetes Mellitus and Heredity | 15 |
Chapter 3 athophysiology of Diabetes Mellitus | 27 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormalities action activity agents antibodies associated betes blood glucose blood glucose concentrations body carbohydrate cause cells changes Chapter clinical complications continuous daily decrease determinations diabetes mellitus diabetic diet diabetic ketoacidosis diagnosis diet disease dose early effect exercise factors Figure given glucose tolerance glycemic glycosylated hemoglobin hormone human insulin hypoglycemia impaired important improved increased indicated individuals initial injection insulin infusion insulin receptor insulin resistance insulin secretion insulin therapy insulin-dependent levels lipoprotein liver meals measure metabolic methods mg/dl mmol/L monitoring muscle myocardial infarction needed normal obese occur oral patients with diabetes patients with Type plasma glucose pregnancy prevent production protein pump receptor reduced regular renal reported response retinopathy risk severe Source studies subcutaneous subjects Table therapy tion tissue treated treatment Type I diabetes Type II units urine usually values weight