Diabetes MellitusJohn A. Galloway, Janet H. Potvin, Charles R. Shuman Lilly Research Laboratories, 1988 - 335 pages |
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Page 68
... ( lipoprotein lipase ) Endothelial wall LCAT ( lecithin cholesterol HDL acyl transferase ) Table 5.2 . Function ... lipoprotein ( HDL ) , which is lighter than lipoprotein - free plasma . Low - density lipoprotein ( LDL ) is the next ...
... ( lipoprotein lipase ) Endothelial wall LCAT ( lecithin cholesterol HDL acyl transferase ) Table 5.2 . Function ... lipoprotein ( HDL ) , which is lighter than lipoprotein - free plasma . Low - density lipoprotein ( LDL ) is the next ...
Page 72
... LIPOPROTEIN METABOLISM IN DIABETES MELLITUS With its complexities , lipoprotein metabolism is vulnerable to many possible defects that can result in accumulation , deficiency , or abnormal composition of any one of the lipoprotein ...
... LIPOPROTEIN METABOLISM IN DIABETES MELLITUS With its complexities , lipoprotein metabolism is vulnerable to many possible defects that can result in accumulation , deficiency , or abnormal composition of any one of the lipoprotein ...
Page 299
... Lipoprotein Abnormalities in Insulin - Dependent and Noninsulin- Dependent ( Type I and Type II ) Diabetes Mellitus Hypertriglyceridemia ( increased very - low - density lipoprotein , remnant particles , reduced lipoprotein lipase ...
... Lipoprotein Abnormalities in Insulin - Dependent and Noninsulin- Dependent ( Type I and Type II ) Diabetes Mellitus Hypertriglyceridemia ( increased very - low - density lipoprotein , remnant particles , reduced lipoprotein lipase ...
Contents
Definition | 1 |
Chapter 2 iabetes Mellitus and Heredity | 15 |
Chapter 3 athophysiology of Diabetes Mellitus | 27 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormalities action activity agents antibodies associated betes blood glucose blood glucose concentrations body carbohydrate cause cells changes Chapter clinical complications continuous daily decrease determinations diabetes mellitus diabetic diet diabetic ketoacidosis diagnosis diet disease dose early effect exercise factors Figure given glucose tolerance glycemic glycosylated hemoglobin hormone human insulin hypoglycemia impaired important improved increased indicated individuals initial injection insulin infusion insulin receptor insulin resistance insulin secretion insulin therapy insulin-dependent levels lipoprotein liver meals measure metabolic methods mg/dl mmol/L monitoring muscle myocardial infarction needed normal obese occur oral patients with diabetes patients with Type plasma glucose pregnancy prevent production protein pump receptor reduced regular renal reported response retinopathy risk severe Source studies subcutaneous subjects Table therapy tion tissue treated treatment Type I diabetes Type II units urine usually values weight