Diabetes MellitusJohn A. Galloway, Janet H. Potvin, Charles R. Shuman Lilly Research Laboratories, 1988 - 335 pages |
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Page 203
... metabolic acidosis leads to characteristic Kussmaul breath- ing , mental torpor , and eventually to coma . At our center , 80 % of the patients have a clin- ical history of three weeks ' or less duration prior to diagnosis . In some ...
... metabolic acidosis leads to characteristic Kussmaul breath- ing , mental torpor , and eventually to coma . At our center , 80 % of the patients have a clin- ical history of three weeks ' or less duration prior to diagnosis . In some ...
Page 221
... metabolic responses to acute exercise as compared with normal controls . A limited number of studies have been performed to evaluate the effect of exercise training on long- term metabolic control in Type I diabetes . These studies have ...
... metabolic responses to acute exercise as compared with normal controls . A limited number of studies have been performed to evaluate the effect of exercise training on long- term metabolic control in Type I diabetes . These studies have ...
Page 280
... metabolic " no return " V Late The principal determinant is inadequate correction of the metabolic disorder itself ( I ) , acting via hyperglycemia , with or without the associated abnormalities of insulin and other hormones ( e.g. ...
... metabolic " no return " V Late The principal determinant is inadequate correction of the metabolic disorder itself ( I ) , acting via hyperglycemia , with or without the associated abnormalities of insulin and other hormones ( e.g. ...
Contents
Definition | 1 |
Chapter 2 iabetes Mellitus and Heredity | 15 |
Chapter 3 athophysiology of Diabetes Mellitus | 27 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormalities action activity agents antibodies associated betes blood glucose blood glucose concentrations body carbohydrate cause cells changes Chapter clinical complications continuous daily decrease determinations diabetes mellitus diabetic diet diabetic ketoacidosis diagnosis diet disease dose early effect exercise factors Figure given glucose tolerance glycemic glycosylated hemoglobin hormone human insulin hypoglycemia impaired important improved increased indicated individuals initial injection insulin infusion insulin receptor insulin resistance insulin secretion insulin therapy insulin-dependent levels lipoprotein liver meals measure metabolic methods mg/dl mmol/L monitoring muscle myocardial infarction needed normal obese occur oral patients with diabetes patients with Type plasma glucose pregnancy prevent production protein pump receptor reduced regular renal reported response retinopathy risk severe Source studies subcutaneous subjects Table therapy tion tissue treated treatment Type I diabetes Type II units urine usually values weight