Diabetes MellitusJohn A. Galloway, Janet H. Potvin, Charles R. Shuman Lilly Research Laboratories, 1988 - 335 pages |
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Page 32
... muscle . 12 In the postabsorptive state , the net peripheral release of branched chain amino acids from skeletal muscle appears to be the major source of these important compounds for the vascular compartment . In the insulin ...
... muscle . 12 In the postabsorptive state , the net peripheral release of branched chain amino acids from skeletal muscle appears to be the major source of these important compounds for the vascular compartment . In the insulin ...
Page 39
... muscle , since it contracts at a high rate during sustained exercise . Because of insulin deficiency , lipolysis of tri- glycerides is accelerated in both adipose and muscle tissue . Intracellular free fatty acid is increased in muscle ...
... muscle , since it contracts at a high rate during sustained exercise . Because of insulin deficiency , lipolysis of tri- glycerides is accelerated in both adipose and muscle tissue . Intracellular free fatty acid is increased in muscle ...
Page 144
... muscle , 7.10 with only minimal amounts of infused glucose utilized by adipose tissue . 44,45 Difficulties in isolating intact muscle cells and the presence in muscle of proteolytic enzymes that degrade both insulin and insulin ...
... muscle , 7.10 with only minimal amounts of infused glucose utilized by adipose tissue . 44,45 Difficulties in isolating intact muscle cells and the presence in muscle of proteolytic enzymes that degrade both insulin and insulin ...
Contents
Definition | 1 |
Chapter 2 iabetes Mellitus and Heredity | 15 |
Chapter 3 athophysiology of Diabetes Mellitus | 27 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormalities action activity agents antibodies associated betes blood glucose blood glucose concentrations body carbohydrate cause cells changes Chapter clinical complications continuous daily decrease determinations diabetes mellitus diabetic diet diabetic ketoacidosis diagnosis diet disease dose early effect exercise factors Figure given glucose tolerance glycemic glycosylated hemoglobin hormone human insulin hypoglycemia impaired important improved increased indicated individuals initial injection insulin infusion insulin receptor insulin resistance insulin secretion insulin therapy insulin-dependent levels lipoprotein liver meals measure metabolic methods mg/dl mmol/L monitoring muscle myocardial infarction needed normal obese occur oral patients with diabetes patients with Type plasma glucose pregnancy prevent production protein pump receptor reduced regular renal reported response retinopathy risk severe Source studies subcutaneous subjects Table therapy tion tissue treated treatment Type I diabetes Type II units urine usually values weight